Was lack of C9ORF72 protein to blame for this vulnerability? To test this, the authors upped C9ORF72 expression in patient and control iMNs by expressing the normal gene off a retrovirus. This rescued survival in patient iMNs and had no effect on controls. Then, the researchers knocked out one or both ...
from Google Alert - vulnerability http://ift.tt/2Bl8WKD
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